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Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair

Само за регистроване кориснике
2019
Аутори
Radak, Đorđe J.
Nešković, Mihailo
Otašević, Petar
Isenović, Esma R.
Чланак у часопису (Рецензирана верзија)
Метаподаци
Приказ свих података о документу
Апстракт
Abdominal Aortic Aneurysm (AAA) is a degenerative disease of the aortic wall with potentially fatal complications. Open Repair (OR) was considered the gold standard, until the emergence of Endovascular Aneurysm Repair (EVAR), which is less invasive and equally (if not more) effective. As the popularity of endovascular procedures grows, related complications become more evident, with kidney damage being one of them. Although Acute Kidney Injury (AKI) following EVAR is relatively common, its true incidence is still uncertain. Also, there is insufficient data concerning long-term renal outcomes after EVAR, especially with repeated contrast agent exposure. Despite the lack of firm evidence on the effectiveness of individual strategies, it is evident that prevention of AKI following EVAR requires a multifactorial approach. This review focuses on recent findings based on human studies regarding the current evidence of renal impairment after EVAR, its quantification and strategies for its pre...vention. © 2019 Bentham Science Publishers.

Кључне речи:
Endovascular aneurysm repair / acute kidney injury / contrast-induced nephropathy / OR / EVAR / Scr
Извор:
Current Vascular Pharmacology, 2019, 17, 2, 133-140
Финансирање / пројекти:
  • Интегрална студија идентификације регионалних генетских фактора ризика и фактора ризика животне средине за масовне незаразне болести хумане популације у Србији - INGEMA_S (RS-MESTD-Integrated and Interdisciplinary Research (IIR or III)-41028)
  • Хормонска регулација експресије и активности азот оксид синтазе и натријум-калијумове пумпе у експерименталним моделима инсулинске резистенције, дијабетеса и кардиоваскуларних поремећаја (RS-MESTD-Basic Research (BR or ON)-173033)
Напомена:
  • This is the peer-reviewed version of the following article: Isenovic, E., D. Radak, M. Neskovic, and P. Otasevic. "Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair." Current vascular pharmacology (2017). http://dx.doi.org/10.2174/1570161115666171116163203
  • Published version: https://vinar.vin.bg.ac.rs/handle/123456789/8038

DOI: 10.2174/1570161115666171116163203

ISSN: 1570-1611

PubMed: 29149818

WoS: 000459678000007

Scopus: 2-s2.0-85060143176
[ Google Scholar ]
7
5
URI
https://vinar.vin.bg.ac.rs/handle/123456789/8040
Колекције
  • Radovi istraživača
Институција/група
Vinča
TY  - JOUR
AU  - Radak, Đorđe J.
AU  - Nešković, Mihailo
AU  - Otašević, Petar
AU  - Isenović, Esma R.
PY  - 2019
UR  - https://vinar.vin.bg.ac.rs/handle/123456789/8040
AB  - Abdominal Aortic Aneurysm (AAA) is a degenerative disease of the aortic wall with potentially fatal complications. Open Repair (OR) was considered the gold standard, until the emergence of Endovascular Aneurysm Repair (EVAR), which is less invasive and equally (if not more) effective. As the popularity of endovascular procedures grows, related complications become more evident, with kidney damage being one of them. Although Acute Kidney Injury (AKI) following EVAR is relatively common, its true incidence is still uncertain. Also, there is insufficient data concerning long-term renal outcomes after EVAR, especially with repeated contrast agent exposure. Despite the lack of firm evidence on the effectiveness of individual strategies, it is evident that prevention of AKI following EVAR requires a multifactorial approach. This review focuses on recent findings based on human studies regarding the current evidence of renal impairment after EVAR, its quantification and strategies for its prevention. © 2019 Bentham Science Publishers.
T2  - Current Vascular Pharmacology
T1  - Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair
VL  - 17
IS  - 2
SP  - 133
EP  - 140
DO  - 10.2174/1570161115666171116163203
ER  - 
@article{
author = "Radak, Đorđe J. and Nešković, Mihailo and Otašević, Petar and Isenović, Esma R.",
year = "2019",
abstract = "Abdominal Aortic Aneurysm (AAA) is a degenerative disease of the aortic wall with potentially fatal complications. Open Repair (OR) was considered the gold standard, until the emergence of Endovascular Aneurysm Repair (EVAR), which is less invasive and equally (if not more) effective. As the popularity of endovascular procedures grows, related complications become more evident, with kidney damage being one of them. Although Acute Kidney Injury (AKI) following EVAR is relatively common, its true incidence is still uncertain. Also, there is insufficient data concerning long-term renal outcomes after EVAR, especially with repeated contrast agent exposure. Despite the lack of firm evidence on the effectiveness of individual strategies, it is evident that prevention of AKI following EVAR requires a multifactorial approach. This review focuses on recent findings based on human studies regarding the current evidence of renal impairment after EVAR, its quantification and strategies for its prevention. © 2019 Bentham Science Publishers.",
journal = "Current Vascular Pharmacology",
title = "Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair",
volume = "17",
number = "2",
pages = "133-140",
doi = "10.2174/1570161115666171116163203"
}
Radak, Đ. J., Nešković, M., Otašević, P.,& Isenović, E. R.. (2019). Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair. in Current Vascular Pharmacology, 17(2), 133-140.
https://doi.org/10.2174/1570161115666171116163203
Radak ĐJ, Nešković M, Otašević P, Isenović ER. Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair. in Current Vascular Pharmacology. 2019;17(2):133-140.
doi:10.2174/1570161115666171116163203 .
Radak, Đorđe J., Nešković, Mihailo, Otašević, Petar, Isenović, Esma R., "Renal Dysfunction Following Elective Endovascular Aortic Aneurysm Repair" in Current Vascular Pharmacology, 17, no. 2 (2019):133-140,
https://doi.org/10.2174/1570161115666171116163203 . .

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