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dc.creatorTrpković, Andreja
dc.creatorStokić, Edita
dc.creatorRadak, Đorđe J.
dc.creatorGluvić, Zoran
dc.creatorHaidara, Mohamed A.
dc.creatorMikhailidis, Dimitri P.
dc.creatorIsenović, Esma R.
dc.date.accessioned2018-03-01T21:34:53Z
dc.date.available2018-03-01T21:34:53Z
dc.date.issued2010
dc.identifier.issn1381-6128 (print)
dc.identifier.issn1873-4286 (electronic)
dc.identifier.urihttp://vinar.vin.bg.ac.rs/handle/123456789/4208
dc.description.abstractThe role of hepatitis C virus (HCV) infection in the development of vascular disease is controversial. Insulin resistance (IR) is a recognized risk factor for cardiovascular disease (CVD) and is associated with chronic hepatitis C (CHC). Thus, IR may promote atherosclerosis and vascular disease in CHC patients. HCV-associated IR may also cause hepatic steatosis and resistance to antiviral treatment. In addition, HCV may contribute a direct, proatherogenetic action on the vascular wall. This review considers the impact of IR on interferon-based therapy of HCV infection and the role of insulin-sensitizing agents on the response to antiviral treatment and prevention of IR complications, including CVD.en
dc.relationinfo:eu-repo/grantAgreement/MESTD/Basic Research (BR or ON)/173033/RS//
dc.rightsrestrictedAccessen
dc.sourceCurrent Pharmaceutical Designen
dc.subjectHepatitis Cen
dc.subjectinsulin resistanceen
dc.subjectinterferon-alphaen
dc.subjectvascular diseaseen
dc.titleChronic Hepatitis C, Insulin Resistance and Vascular Diseaseen
dc.typereviewen
dcterms.abstractРадак, Дјордје; Исеновић Есма; Глувиц, Зоран; Хаидара, Мохамед; Микхаилидис, Димитри П.; Трпковиц, Aндреја; Стокиц, Едитх;
dc.citation.volume16
dc.citation.issue34
dc.citation.spage3823
dc.citation.epage3829
dc.identifier.wos000286877500006
dc.identifier.doi10.2174/138161210794455067
dc.citation.rankM21a
dc.identifier.pmid21128893
dc.identifier.scopus2-s2.0-79551635641


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