Patofiziologija gojaznosti
Pathophysiology of obesity
Нема приказа
Аутори
Dobutović, BranislavaSudar, Emina
Soskić, Sanja S.
Obradović, Milan M.
Nikolić, Dragana
Gluvić, Zoran
Stokić, Edita
Radak, Đorđe J.
Isenović, Esma R.
Чланак у часопису (Објављена верзија)
Метаподаци
Приказ свих података о документуАпстракт
Gojaznost se danas smatra bolešću savremenog društva i njena zastupljenost u svetu ima karakter epidemije. Istraživanja usmerena na razumevanje biologije adipocitne ćelije i adipoznog tkiva, značajno doprinose rasvetljavanju mnogih aspekata različitih metaboličkih poremećaja koji prate pojavu gojaznosti tj. prekomerenog prisustva adipoznog tkiva. Gojaznost je posledica dugotrajnog pozitivnog energetskog balansa, neprimerenog stilu života savremenog čoveka. Danas je utemeljena činjenica da je adipozno tkivo endokrini organ u kojem se sintetiše i eksprimira više od 60 različitih faktora - adipokina, sa snažnim uticajem na veliki broj metaboličkih procesa u organizmu. Adipokini mogu imati pro-inflamatorno ili anti-inflamatorno dejstvo. Mnogobrojna istraživanja jasno ukazuju na uzročnu vezu gojaznosti i hronične inflamacije slabog inteziteta, koja vodi razvoju poremećaja vezanih za prisustvo gojaznosti, naročito metaboličkih poremećaja koji znatno umanjuju kvalitet života. Svojom endokrino...m funkcijom, adipocitne ćelije reflektuju metabolički status i prenose informaciju na druge organe, tkiva kao i centralni nervni sistem.
Obesity is a disease of the modern society and considering the number of obese people it has character of the worldwide epidemic. Understanding the biology of adipocytes and the events occurring in adipose tissue contributes to clarification of many aspects of the various metabolic disorders associated with obesity and excessive presence of adipose tissue. Overweight in individuals is the result of a long-term positive energy balance inappropriate to life style of the modern man. Today, it is well accepted that adipose tissue is an endocrine organ in which more than 60 of adipokines are synthesized and are expressed with strong influence on a great number of metabolic processes in organism. Adipokines have pro-inflammatory or anti-inflammatory effects. Increasing evidence indicates that obesity is causally linked to a chronic low-grade inflammatory state, and contributes to the development of obesity-linked disorders, in particular to metabolic dysfunction which significantly reduces t...he quality of life. Adipocytes, with their endocrine function, reflect metabolic status and transport information into organs, tissues and central nervous system, too.
Кључне речи:
gojaznost / adipokini / rezistin / adiponektin / leptin / obesity / adipokines / resistin / adiponectin / leptinИзвор:
Medicinska istraživanja, 2012, 46, 1, 43-54Колекције
Институција/група
VinčaTY - JOUR AU - Dobutović, Branislava AU - Sudar, Emina AU - Soskić, Sanja S. AU - Obradović, Milan M. AU - Nikolić, Dragana AU - Gluvić, Zoran AU - Stokić, Edita AU - Radak, Đorđe J. AU - Isenović, Esma R. PY - 2012 UR - https://vinar.vin.bg.ac.rs/handle/123456789/10323 AB - Gojaznost se danas smatra bolešću savremenog društva i njena zastupljenost u svetu ima karakter epidemije. Istraživanja usmerena na razumevanje biologije adipocitne ćelije i adipoznog tkiva, značajno doprinose rasvetljavanju mnogih aspekata različitih metaboličkih poremećaja koji prate pojavu gojaznosti tj. prekomerenog prisustva adipoznog tkiva. Gojaznost je posledica dugotrajnog pozitivnog energetskog balansa, neprimerenog stilu života savremenog čoveka. Danas je utemeljena činjenica da je adipozno tkivo endokrini organ u kojem se sintetiše i eksprimira više od 60 različitih faktora - adipokina, sa snažnim uticajem na veliki broj metaboličkih procesa u organizmu. Adipokini mogu imati pro-inflamatorno ili anti-inflamatorno dejstvo. Mnogobrojna istraživanja jasno ukazuju na uzročnu vezu gojaznosti i hronične inflamacije slabog inteziteta, koja vodi razvoju poremećaja vezanih za prisustvo gojaznosti, naročito metaboličkih poremećaja koji znatno umanjuju kvalitet života. Svojom endokrinom funkcijom, adipocitne ćelije reflektuju metabolički status i prenose informaciju na druge organe, tkiva kao i centralni nervni sistem. AB - Obesity is a disease of the modern society and considering the number of obese people it has character of the worldwide epidemic. Understanding the biology of adipocytes and the events occurring in adipose tissue contributes to clarification of many aspects of the various metabolic disorders associated with obesity and excessive presence of adipose tissue. Overweight in individuals is the result of a long-term positive energy balance inappropriate to life style of the modern man. Today, it is well accepted that adipose tissue is an endocrine organ in which more than 60 of adipokines are synthesized and are expressed with strong influence on a great number of metabolic processes in organism. Adipokines have pro-inflammatory or anti-inflammatory effects. Increasing evidence indicates that obesity is causally linked to a chronic low-grade inflammatory state, and contributes to the development of obesity-linked disorders, in particular to metabolic dysfunction which significantly reduces the quality of life. Adipocytes, with their endocrine function, reflect metabolic status and transport information into organs, tissues and central nervous system, too. T2 - Medicinska istraživanja T1 - Patofiziologija gojaznosti T1 - Pathophysiology of obesity VL - 46 IS - 1 SP - 43 EP - 54 UR - https://hdl.handle.net/21.15107/rcub_vinar_10323 ER -
@article{ author = "Dobutović, Branislava and Sudar, Emina and Soskić, Sanja S. and Obradović, Milan M. and Nikolić, Dragana and Gluvić, Zoran and Stokić, Edita and Radak, Đorđe J. and Isenović, Esma R.", year = "2012", abstract = "Gojaznost se danas smatra bolešću savremenog društva i njena zastupljenost u svetu ima karakter epidemije. Istraživanja usmerena na razumevanje biologije adipocitne ćelije i adipoznog tkiva, značajno doprinose rasvetljavanju mnogih aspekata različitih metaboličkih poremećaja koji prate pojavu gojaznosti tj. prekomerenog prisustva adipoznog tkiva. Gojaznost je posledica dugotrajnog pozitivnog energetskog balansa, neprimerenog stilu života savremenog čoveka. Danas je utemeljena činjenica da je adipozno tkivo endokrini organ u kojem se sintetiše i eksprimira više od 60 različitih faktora - adipokina, sa snažnim uticajem na veliki broj metaboličkih procesa u organizmu. Adipokini mogu imati pro-inflamatorno ili anti-inflamatorno dejstvo. Mnogobrojna istraživanja jasno ukazuju na uzročnu vezu gojaznosti i hronične inflamacije slabog inteziteta, koja vodi razvoju poremećaja vezanih za prisustvo gojaznosti, naročito metaboličkih poremećaja koji znatno umanjuju kvalitet života. Svojom endokrinom funkcijom, adipocitne ćelije reflektuju metabolički status i prenose informaciju na druge organe, tkiva kao i centralni nervni sistem., Obesity is a disease of the modern society and considering the number of obese people it has character of the worldwide epidemic. Understanding the biology of adipocytes and the events occurring in adipose tissue contributes to clarification of many aspects of the various metabolic disorders associated with obesity and excessive presence of adipose tissue. Overweight in individuals is the result of a long-term positive energy balance inappropriate to life style of the modern man. Today, it is well accepted that adipose tissue is an endocrine organ in which more than 60 of adipokines are synthesized and are expressed with strong influence on a great number of metabolic processes in organism. Adipokines have pro-inflammatory or anti-inflammatory effects. Increasing evidence indicates that obesity is causally linked to a chronic low-grade inflammatory state, and contributes to the development of obesity-linked disorders, in particular to metabolic dysfunction which significantly reduces the quality of life. Adipocytes, with their endocrine function, reflect metabolic status and transport information into organs, tissues and central nervous system, too.", journal = "Medicinska istraživanja", title = "Patofiziologija gojaznosti, Pathophysiology of obesity", volume = "46", number = "1", pages = "43-54", url = "https://hdl.handle.net/21.15107/rcub_vinar_10323" }
Dobutović, B., Sudar, E., Soskić, S. S., Obradović, M. M., Nikolić, D., Gluvić, Z., Stokić, E., Radak, Đ. J.,& Isenović, E. R.. (2012). Patofiziologija gojaznosti. in Medicinska istraživanja, 46(1), 43-54. https://hdl.handle.net/21.15107/rcub_vinar_10323
Dobutović B, Sudar E, Soskić SS, Obradović MM, Nikolić D, Gluvić Z, Stokić E, Radak ĐJ, Isenović ER. Patofiziologija gojaznosti. in Medicinska istraživanja. 2012;46(1):43-54. https://hdl.handle.net/21.15107/rcub_vinar_10323 .
Dobutović, Branislava, Sudar, Emina, Soskić, Sanja S., Obradović, Milan M., Nikolić, Dragana, Gluvić, Zoran, Stokić, Edita, Radak, Đorđe J., Isenović, Esma R., "Patofiziologija gojaznosti" in Medicinska istraživanja, 46, no. 1 (2012):43-54, https://hdl.handle.net/21.15107/rcub_vinar_10323 .