TY  - JOUR
AU  - Bin-Jaliah, Ismaeel
AU  - Ammar, Hania I.
AU  - Mikhailidis, Dimitri P.
AU  - Dallak, Mohammed A.
AU  - Al-Hashem, Fahaid H.
AU  - Haidara, Mohamed A.
AU  - Yassin, Hanaa Z.
AU  - Bahnasi, Abeer A.
AU  - Rashed, Laila A.
AU  - Isenović, Esma R.
PY  - 2010
UR  - https://vinar.vin.bg.ac.rs/handle/123456789/3883
AB  - The role of vascular endothelial growth factor (VEGF) and erythropoietin (EPO) in mediating hypoxic preconditioning under the acute intermittent hypoxic condition (AIH) was investigated in this study. Male Wistar rats were randomly assigned and kept in normoxic conditions, (Nx) or in AIH conditions and subjected to brief cycles hypoxia/reoxygenation. Hearts were isolated, perfused, and subjected to in vitro global ischemia followed by reperfusion. During and at the end of reperfusion, left ventricular developed pressure (LVDP); LV end diastolic pressure (LVEDP); rate pressure product (RPP); peak left ventricular pressure rise (Delta P/Delta t(max)) and heart rate (HR) were measured. Hearts subjected to AIH displayed a significant higher LVDP (P LT .001), RPP (P LT .001), and Delta P/Delta t(max) (P LT .001). Expression of VEGF and EPO were significantly increased at 3, 8, and 24 hours after AIH. Hypoxic training could provide a new approach to enhance endogenous cardioprotective mechanisms.
T2  - Angiology
T1  - Cardiac Adaptive Responses After Hypoxia in an Experimental Model
VL  - 61
IS  - 2
SP  - 145
EP  - 156
DO  - 10.1177/0003319709352486
ER  - 

@article{
author = "Bin-Jaliah, Ismaeel and Ammar, Hania I. and Mikhailidis, Dimitri P. and Dallak, Mohammed A. and Al-Hashem, Fahaid H. and Haidara, Mohamed A. and Yassin, Hanaa Z. and Bahnasi, Abeer A. and Rashed, Laila A. and Isenović, Esma R.",
year = "2010",
abstract = "The role of vascular endothelial growth factor (VEGF) and erythropoietin (EPO) in mediating hypoxic preconditioning under the acute intermittent hypoxic condition (AIH) was investigated in this study. Male Wistar rats were randomly assigned and kept in normoxic conditions, (Nx) or in AIH conditions and subjected to brief cycles hypoxia/reoxygenation. Hearts were isolated, perfused, and subjected to in vitro global ischemia followed by reperfusion. During and at the end of reperfusion, left ventricular developed pressure (LVDP); LV end diastolic pressure (LVEDP); rate pressure product (RPP); peak left ventricular pressure rise (Delta P/Delta t(max)) and heart rate (HR) were measured. Hearts subjected to AIH displayed a significant higher LVDP (P LT .001), RPP (P LT .001), and Delta P/Delta t(max) (P LT .001). Expression of VEGF and EPO were significantly increased at 3, 8, and 24 hours after AIH. Hypoxic training could provide a new approach to enhance endogenous cardioprotective mechanisms.",
journal = "Angiology",
title = "Cardiac Adaptive Responses After Hypoxia in an Experimental Model",
volume = "61",
number = "2",
pages = "145-156",
doi = "10.1177/0003319709352486"
}

Bin-Jaliah, I., Ammar, H. I., Mikhailidis, D. P., Dallak, M. A., Al-Hashem, F. H., Haidara, M. A., Yassin, H. Z., Bahnasi, A. A., Rashed, L. A.,& Isenović, E. R.. (2010). Cardiac Adaptive Responses After Hypoxia in an Experimental Model. in Angiology, 61(2), 145-156.
https://doi.org/10.1177/0003319709352486

Bin-Jaliah I, Ammar HI, Mikhailidis DP, Dallak MA, Al-Hashem FH, Haidara MA, Yassin HZ, Bahnasi AA, Rashed LA, Isenović ER. Cardiac Adaptive Responses After Hypoxia in an Experimental Model. in Angiology. 2010;61(2):145-156.
doi:10.1177/0003319709352486 .

Bin-Jaliah, Ismaeel, Ammar, Hania I., Mikhailidis, Dimitri P., Dallak, Mohammed A., Al-Hashem, Fahaid H., Haidara, Mohamed A., Yassin, Hanaa Z., Bahnasi, Abeer A., Rashed, Laila A., Isenović, Esma R., "Cardiac Adaptive Responses After Hypoxia in an Experimental Model" in Angiology, 61, no. 2 (2010):145-156,
https://doi.org/10.1177/0003319709352486 . .

TY  - JOUR
AU  - Fouad, Hanan H.
AU  - Al-Dera, Husain
AU  - Bakhoum, Sameh W.
AU  - Rashed, Laila A.
AU  - Sayed, Rehab H.
AU  - Rateb, Moshira A.
AU  - Haidara, Mohamed A.
AU  - Soskić, Sanja S.
AU  - Isenović, Esma R.
PY  - 2010
UR  - https://vinar.vin.bg.ac.rs/handle/123456789/4039
AB  - We determined the serum levels of soluble CD40 ligand (sCD4OL) in patients with chronic coronary artery disease (CAD) and acute coronary syndrome (ACS). Patients with unstable angina (UA) and myocardial infarction (MI) showed significantly higher levels (P LT .001) of sCD4OL compared with patients with stable angina (SA) and controls; particularly, high levels occurred in patients with UA (UA: 9.23 +/- 2.92, MI: 7.38 +/- 1.05, SA: 4.42 +/- 1.08; control: 4.01 +/- 0.87 ng/mL). There was no significant difference in sCD4OL levels between patients with UA and MI or between patients with SA and controls. Levels of sCD4OL did not show any significant correlation with peak creatine kinase (CK), CK-MB isoenzyme activity in patients with MI, troponin T serum levels in patients with UA or with culprit vessel (CV) complexity score (CVCS), type of CV lesion, or vessel score in patients with UA or MI. These results suggest that CD40L plays a pathogenic role in triggering ACS.
T2  - Angiology
T1  - Levels of sCD40 Ligand in Chronic and Acute Coronary Syndromes and its Relation to Angiographic Extent of Coronary Arterial Narrowing
VL  - 61
IS  - 6
SP  - 567
EP  - 573
DO  - 10.1177/0003319709356785
ER  - 

@article{
author = "Fouad, Hanan H. and Al-Dera, Husain and Bakhoum, Sameh W. and Rashed, Laila A. and Sayed, Rehab H. and Rateb, Moshira A. and Haidara, Mohamed A. and Soskić, Sanja S. and Isenović, Esma R.",
year = "2010",
abstract = "We determined the serum levels of soluble CD40 ligand (sCD4OL) in patients with chronic coronary artery disease (CAD) and acute coronary syndrome (ACS). Patients with unstable angina (UA) and myocardial infarction (MI) showed significantly higher levels (P LT .001) of sCD4OL compared with patients with stable angina (SA) and controls; particularly, high levels occurred in patients with UA (UA: 9.23 +/- 2.92, MI: 7.38 +/- 1.05, SA: 4.42 +/- 1.08; control: 4.01 +/- 0.87 ng/mL). There was no significant difference in sCD4OL levels between patients with UA and MI or between patients with SA and controls. Levels of sCD4OL did not show any significant correlation with peak creatine kinase (CK), CK-MB isoenzyme activity in patients with MI, troponin T serum levels in patients with UA or with culprit vessel (CV) complexity score (CVCS), type of CV lesion, or vessel score in patients with UA or MI. These results suggest that CD40L plays a pathogenic role in triggering ACS.",
journal = "Angiology",
title = "Levels of sCD40 Ligand in Chronic and Acute Coronary Syndromes and its Relation to Angiographic Extent of Coronary Arterial Narrowing",
volume = "61",
number = "6",
pages = "567-573",
doi = "10.1177/0003319709356785"
}

Fouad, H. H., Al-Dera, H., Bakhoum, S. W., Rashed, L. A., Sayed, R. H., Rateb, M. A., Haidara, M. A., Soskić, S. S.,& Isenović, E. R.. (2010). Levels of sCD40 Ligand in Chronic and Acute Coronary Syndromes and its Relation to Angiographic Extent of Coronary Arterial Narrowing. in Angiology, 61(6), 567-573.
https://doi.org/10.1177/0003319709356785

Fouad HH, Al-Dera H, Bakhoum SW, Rashed LA, Sayed RH, Rateb MA, Haidara MA, Soskić SS, Isenović ER. Levels of sCD40 Ligand in Chronic and Acute Coronary Syndromes and its Relation to Angiographic Extent of Coronary Arterial Narrowing. in Angiology. 2010;61(6):567-573.
doi:10.1177/0003319709356785 .

Fouad, Hanan H., Al-Dera, Husain, Bakhoum, Sameh W., Rashed, Laila A., Sayed, Rehab H., Rateb, Moshira A., Haidara, Mohamed A., Soskić, Sanja S., Isenović, Esma R., "Levels of sCD40 Ligand in Chronic and Acute Coronary Syndromes and its Relation to Angiographic Extent of Coronary Arterial Narrowing" in Angiology, 61, no. 6 (2010):567-573,
https://doi.org/10.1177/0003319709356785 . .